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Reaction catalyzed by HGPRT is clinically relevant as deficiencies can cause accumulation of uric acid

Not all trials quantitatively specify the bioactive content of the intervention material

To identify the protein partners involved in this regulatory mechanism, we analyzed the proteins enriched by the LTFe probes

Along with matrix effects and multiple variable experimental conditions, performing the assays (radical generator, time of measure, expression of results, etc.) makes it difficult to compare the reported data

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We then investigated whether this protective effect was due to increased intracellular GSH levels