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To validate that dystroglycanlaminin-211 interaction is the predominant mechanism mediating melanocyte adhesion in healthy skin, we used antibody blockade, knockdown by small interfering RNA (siRNA), and enzymatic deglycosylation approaches

These metabolic changes can limit the work capacity of skeletal muscle, since the accumulated acetyl-CoA inhibits the activity of pyruvate dehydrogenase [64]

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