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Methylselenic acid (MSeA) has been observed to augment caspase-mediated apoptosis by down-regulating the expression of survivin, Bcl-xL, and Mcl-1, which are proteins that typically inhibit apoptosis (144, 145)

However, in Nqo1 and Slc7a11 loci, only ARE motifs are predicted while no CpG island was identified in 15 kb surrounding the transcription start site of both genes (Supplementary Fig

Protective Role of Glutathione in the Hippocampus after Brain Ischemia

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