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doi: 10.1093/jnci/djt181
As previously mentioned, CPT1 may be allosterically inhibited by malonyl-CoA, a product of ACC (Figure 3)
Unbiased transcriptomic and metabolomic analyses showed that chronic 2 -adrenergic receptor stimulation caused metabolic reprogramming of skeletal muscle characterized by enhanced glucose utilization
European Association of the Study of the Liver
Am J Clin Nutr 100:778786
VPA was stopped and L-carnitine supplementation (100 mg/kg) was administered intravenously
