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MHCII Is Required for -Synuclein-Induced Activation of Microglia, CD4 T Cell Proliferation, and Dopaminergic Neurodegeneration

Importantly, these effects are highly strain-specific and in some cases, dependent on the viability of the organisms, their metabolites, or cell surface components

elegans models, XBP1s mitigates amyloid- or tau toxicity

Several factors can enhance neuroplasticity, including enriched environments, physical exercise, and pharmacological treatments such as selective serotonin reuptake inhibitors (SSRIs), N-methyl-D-aspartate (NMDA) receptor modulators like ketamine, and neurotrophic agents such as BDNF enhancers

Kennedy, S

Indian J Gastroenterol 42, 448451 (2023) What are the possible risks or side effects