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Using the pressure dependent model, we can therefore hypothesize that high levels of ApoC-II on the TRL surface would increase the surface pressure preventing LPL binding, thereby attenuating lipolysis, when ApoC-II is overexpressed

Cholesteryl esters are transferred back to the remnant particles in exchange for triglycerides by cholesteryl ester transport protein (CETP)

Statistical analyses were conducted using SPSS Statistics version 21 (IBM, New York, USA)

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