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Next, we determined how ALS-linked mutations in FUS/TLS affected association with U1-snRNP components, using isogenic cell lines expressing a single copy of LAP-tagged wild-type and ALS-linked FUS/TLS mutations R495X, R514G, R521G and P525L

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IL-33 promotes an innate immune pathway of intestinal tissue protection dependent on amphiregulinEGFR interactions

reports research funding, advisory/consulting fees and/or other support from AstraZeneca, Bausch Health Inc., Boehringer Ingelheim, CIHR, Janssen, Eli Lilly & Company and Novo Nordisk

(1998b)

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