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Mutation, deletion or inactivation of the gene that encodes ALS in humans and mice decreases circulating levels of IGF-1 and IGFBP-3, causing moderate growth deficiency and abnormalities in bone and carbohydrate metabolism
[48] Martina V, et al
The mice were treated daily with BSO following the approved animal experiment protocol
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Therefore, future research efforts should be directed toward developing approaches that modulate this network signaling system as a cohesive unit
