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In vitro investigations have indicated that ALA has neuroprotective effects on A-mediated cytotoxicity [54,55,56], namely through defending cortical neurons from cytotoxicity induced by A or H 2 O 2 [57], partially attributed to the activation of PKB/Akt signaling pathway

360 At the cellular level, excess of ammonium mainly affects astrocytes with neuronal injury being secondary, resulting mainly from failure of astrocytic homoeostatic cascades and oedema

Front Bioeng Biotechnol 9:670124

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