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The prospect of epigenetics-based personalized medicine for MS patients is interesting, but the number of epigenetic changes that any one individual may have must be taken into account

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Later, as research continued, it was demonstrated to stimulate growth and differentiation of a number of cell lines [6,7,8,9,10] and act as a growth inhibitor for some cultured cells [11], establishing itself as a modulating peptide [12]

The de-S-nitrosylation of G6PD could be a compensatory response to decreased NO and increased ROS generation during EC senescence