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Using advanced tracer methodologies, the researchers identified a fascinating metabolic phenomenon they termed "brain glucose steal." Due to systemic mitochondrial impairment, non-brain organs were essentially stealing glucose intended for the central nervous system, starving the brain of energy and driving cognitive dysfunction

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49,50 These findings validate MAOB as a promising therapeutic target, consistent with our findings that MAOB-mediated astrocytic GABA synthesis contributes to PTSD pathophysiology

J Sports Sci 35:21852190

Role of antioxidant systems, lipid peroxidation, and nitric oxide in postmenopausal osteoporosis

Semiquantitatively evaluated intensity of (a) hemorrhagic zone, (b) edema, and (c) loss of neurons in anterior horn and intermediate gray matter of spinal cord (0no changes