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Thus, the contribution of high-fructose intake for the concurrent development on NASH and atherosclerosis warrants further research

[DOI] [PMC free article] [PubMed] [Google Scholar] [48].Yin Z, Ivanov V, Habelhah H, Tew KD, Ronai Z

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Although it is still unclear how necrosis can decrease overall survival in both pre-clinical and clinical GBMs, we suspect that secretion of pro-tumorigenic factors and/or recruitment and activation of other immune cells (e.g., microglia) by ferroptosed tumor cells may lead to certain non-autonomous tumor effects that precipitate neuroinflammation (or, more precisely, necroinflammation) and cerebral cytokine storm, eventually leading to irreversible cerebral edema, cachexia, multi-organ dysfunction, and death, as observed in our GBM mouse model

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