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Mansur et al ., 2022

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doi: 10.1016/s1097-2765(03)00105-9

The exact mechanism of eNOS modification is unknown but in vivo experiments have shown that eNOS activation in aortas and iNOS transgenetic expression in mouse heart both result in NO-induced protein S-glutathionylation

Studies examining the relationship between caffeine consumption and all-cause and cause-specific mortality have yielded intriguing findings, suggesting both protective and neutral effects

Role of endoplasmic reticulum stress in the loss of retinal ganglion cells in diabetic retinopathy