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In addition, it can be suggested that the elevated glutamate uptake capacity in the astrocytes might be in response to increased glutamate shuttling form neurons which possibly release the excess glutamate that is not incorporated in the TCA cycle due to hampered GDH levels

M.ReisM

Overall, the position of different mutations can affect protein catalytic activity and stability to various degrees, with mutations in specific region of the protein leading to more severe deficiency

Mechanisms of cachexia in chronic disease states

Antioxidant imbalance in the lungs of cystic fibrosis transmembrane conductance regulator protein mutant mice

Still, its role in relieving Sub stress has yet to be explored