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Our experiments in mouse models of mild hyperhomocysteinemia caused by a deficiency in cystathionine beta-synthase in CBS +/ (heterozygous deficient) mice found that increased plasma total homocysteine caused abnormal responses to endothelium-dependent vasodilators

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In an experimental model of induced fatigue bone damage, administration of CGRP or CGRP(837) increased reparative bone formation [74]

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