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[PubMed: 1569148] 96

MUC1 and HIF-1alpha signaling crosstalk induces anabolic glucose metabolism to impart gemcitabine resistance to pancreatic cancer [published correction appears in cancer cell

DCFH-DA probe for LPS-induced ROS Raw 264.7 cells were seeded into a 12-well plate and stimulated with LPS for 24 h to differentiate into inflammatory cells

Alteration of such ratio during oxidative burst can modulate redox signaling pathways related to various cellular processes including proliferation, growth and apoptosis [1]

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Moreover, the endogenous GSH pool is far too low to detoxify exogenous SF completely suggesting that reversible GSH conjugation is an inefficient detoxification mechanism in the presence of high ITC concentrations