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10.1016/S0168-8278(97)80186-9 [DOI] [PubMed] [Google Scholar] 83.Keller TL, Zocco D, Sundrud MS, Hendrick M, Edenius M, Yum J, et al

In addition, there may be potential that these fragments are binding to these same receptors to elicit a zonulin-like effect in tight junction dysregulation, versus an inhibition of zonulin activity, although mechanistic studies need to be conducted to fully explore this possibility

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Increased proteasome-mediated degradation of occludin in irritable bowel syndrome

It seems that the increase in Nrf-2 and its targets HO-1 and MRP-3 might be the specific signature of DR-related Nrf-2 pathway activation