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The paradox is that overusing a pro-collagen peptide destroys collagen through MMP overexpression

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This intricate regulatory network underscores the multifaceted role of FSP1 in modulating the cellular susceptibility to ferroptosis, providing novel insights into the complex interplay between cellular metabolism and iron homeostasis in the context of oxidative stress and cell death

Unresolved inflammation results in formation of vulnerable plaques characterized by enhanced macrophage apoptosis and defective efferocytosis of apoptotic cells resulting in necrotic cell death leading to increased smooth muscle cell death, decreased extracellular matrix production, and collagen degradation by macrophage proteases

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