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doi: 10.1002/biof.1823 275 AlbakerABAlhusainiAMAlbadraniHMSarawiWSAlsabhanJFMansyTWet al
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It, therefore, allows clinicians to appropriately target patient populations to modify their metabolic risk profile to prevent complications [50]
In nonstressed cells the binding of Nrf2 to Keap1 promotes ubiquitination of Nrf2 followed by proteasomal degradation
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