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It seems that metabolic stress during obesity and metabolic syndrome induces a compensatory response, being characterized by increased Zn-mediated mechanisms of antioxidant protection

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Particularly, it determined the upregulation of ROS-scavenging enzymes, anti-inflammatory cytokines, (IL-10, IL-6 and PPAR ), tight junctions proteins, such as ZO-1, MUC-2, claudin-3, and the downregulation of inflammatory genes (TNF-, IL-1) [14]

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The success of GLP-1 agonists in clinical practice has validated the concept that pharmacological approaches to obesity are effective but it has also highlighted their limitations

ALA helps regenerate other antioxidants including vitamin C, vitamin E, and glutathione, potentially extending their functional lifespan and reducing the demand for ongoing synthesis