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A theoretically sound approach based on known mechanisms would combine KPV for NF-kB suppression, thymosin alpha-1 for immune regulation, BPC-157 or GHK-Cu for tissue repair, and collagen peptides for structural support

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This is significant because, if a single valid vector can be identified (see below for criteria), it demonstrates that somatic and affective symptoms have a common core and, hence, a shared pathophysiology [25]
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