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2017 Jun;24(3):8802-8813

This could be directly tied to the observed genome instability in these mutants, due to polyamine roles in increased DNA repair, stability and resistance to oxidative damage, and this pathway is under further investigation by our group

INF39 is a nontoxic, irreversible acrylic ester-induced NLRP3 inflammasome inhibitor that specifically inhibits NLRP3 activation without affecting the NLRC4 or AIM2 inflammasome [184]

Cagrilintide is a longacting acylated analogue of the pancreatic hormone amylin, designed for onceweekly subcutaneous administration [1]

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Even in the absence of infiltration of peripheral inflammatory cells, activation of native CNS cells like microglia are responsible for the progression and maintenance of pathological lesions [36]