glutathione reductase removal of reactive oxygen species, toxicity, oxidative stress, and antioxidants: chronic diseases and aging | Archives Toxicology Mitochondrial Glutathione in Cellular Redox

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Co-transfection experiments revealed that the knockdown of AGO2 in primary hepatocytes abolished the increase of ACSL4 and PRKAA2 expressions induced by miR-214, indicating that miR-214 exerts a positive regulatory function by binding to AGO2 protein in the nucleus

Metabolite profiling reveals the glutathione biosynthetic pathway as a therapeutic target in triple negative breast cancers

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