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Note however that based on our RNA-seq and in situ analyses, the most significant changes in gene expression between control and dMF1/2 mutant mice were observed for Rspo2 and Rspo3 , and thus, their persistent expression in the DP of the dMF1/2 mutant is likely to predominantly contribute to the extended anagen

Thus, defective apoptosis leading to prolonged survival of pathogenic lymphocytes could be one cause of SLE [96]

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