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The most robust theory is that metformin disrupts the B12-intrinsic factor (IF) complex uptake at the ileal cell surface receptor (Cubilin receptor) which is known to be calcium dependent

GLP-1 agonists are better supported for metabolic anti-aging (reducing obesity, inflammation, and cardiovascular risk)

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suggesting that, at high concentrations, IGF-1 has effects not simply mediated by suppressing the effect of GH on hepatic insulin sensitivity [129, 209]

Semaglutide works by stimulating a hormone called glucagon-like peptide-1 (GLP-1) that targets areas of the brain that regulate appetite and food intake