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Although it is unclear how EGFR activation modulates GPx-1 activity, the increase in activity was rapid and peaked within 15 min, suggesting an event that required activation of existing GPx-1, possibly by phosphorylation or through alterations in proteinprotein interactions

For example, wild-type p53 activation in fibrosarcoma cells slows the depletion of glutathione and inhibits xCT activity to prevent ferroptosis induction