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This observation contributes to the expanding body of evidence regarding the potential involvement of selenium in modulating the physiological functions of the adrenal gland (145)

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Voltage-dependent N-type Ca2+ channels in endothelial cells contribute to oxidative stress-related endothelial dysfunction induced by angiotensin II in mice

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Surprisingly, new ahpC suppressor mutations arose in a strain lacking both glutathione and -Glu-Cys, gshA trxB, which ahpC* does not suppress