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10.1186/s12967-015-0477-1 203

detected chemokines in a conditioned medium of AILI-derived hepatocytes revealing the higher abundance of VEGF-D (11-fold), VEGF (1.7-fold), its receptors VEGF-R3 and VEGF-R2 (sixfold), and lower expression of VEGF-R1 (0.7-fold) and HGF-R (0.6-fold), and VEGF may activate the STAT3 and ATM pathways[186]

But in the case of B12 deficiency, homocysteine may rise beyond normal levels, causing irritation to the cardiovascular system and increasing inflammation

The importance of peroxynitrite in mediating APAP-induced liver injury is also illustrated by the fact that direct scavenging of peroxynitrite by mitochondrial GSH49,50 or resveratrol51 also prevented protein nitration and protected against APAP-induced liver injury