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assessed 101 HCV patients and demonstrated a significant correlation between steatosis and HCV RNA level in patients infected with HCV-3, but not in those infected with HCV-1, providing evidence that steatosis is the morphological expression of a viral cytopathic effect of HCV-3.[27] Furthermore, in patients with HCV cirrhosis, genotype 3 has been independently associated with an increased risk of HCC[28] (Figure 1)

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Further highlighting the link between insulin resistance and postprandial lipid abnormalities, a recent study found that insulin resistant, abdominally obese adults had higher postprandial TG concentrations than abdominally obese adults without insulin resistance and non - abdominally obese controls (61, 86)

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The transport reaction catalyzed by ATB 0,+ occurs as a co-transport of 2Na + and 1Cl being highly concentrative and sensitive to membrane potential (Nakanishi et al., 2001)