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Antagonistic action on NMDA/GluN2B mediated currents of two peptides that were conantokin-G structure-based designed
Epithalon monograph final - peptidesociety.org
doi: 10.1016/j.biortech.2010.09.063
J Mol Neurosci MN (2003) 20(2):17388
These effective combination therapies revealed the inherent susceptibility of TNBC to ferroptosis and highlighted the potential strategy of ferroptosis as a drug target of TNBC
