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This review systematically elucidates core pathways through which MPs/NPs drive PD pathology via multidimensional molecular mechanisms: (1) MPs/NPs facilitate -synuclein oligomerization and fibrillation by hydrophobically adsorbing its NAC domain

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IgG or IgM antibodies binding to the erythrocytes leads to immune destruction

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together, these reports suggest that different TNBC subsets may respond quite differently to PRL