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The findings show that UA treatment after stroke leads to the protection of brain endothelial cell function, and this beneficial effect is associated with the decrease in circulating levels of VEGF-A and the increase in KLF2 expression

This cysteine bioavailability decrease could be expected in a situation of the increased requirement of cysteine for the protein synthesis in immune cells

Over-expression of glutathione S -transferase/glutathione peroxidase enhances the growth of Transgenic tobacco seedlings during stress

doi: 10.1002/art.40830

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