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doi: 10.1093/ibd/izab251

Neurochem Res 32:18841890 Scheiber IF, Dringen R (2013) Astrocyte functions in the copper homeostasis of the brain
reported that FMS symptom severity was positively associated with lysophosphatidylcholines (LPCs), suggesting that FMS may be related to altered lipid metabolism (114)

The major pathogenetic mechanism of GD is the stimulation of the thyroid stimulating hormone receptor (TSH-R) by autoantibodies that bind to it and promote thyrocyte proliferation and activity, resulting in thyroid hyperfunction (13)

Higher concentrations aren't necessarily better and may increase irritation risk without proportional benefits

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