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In the last few years, an increasing number of studies have suggested that the m6A modification is involved in the progression of AD and is associated with inflammatory responses [14-16], but specific functional and mechanistic studies have not been reported

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However, only a limited number of studies have been performed to evaluate the toxicity of NSAIDs in the nervous system 15

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A total of 68 articles were included in the final synthesis (corresponding to the reference list), comprising clinical trials and observational studies of GLP-1RA, mechanistic and immunology papers on GLP-1 signaling and metabolic inflammation, literature on autoimmune thyroid disease pathophysiology, adipokines, Non-alcoholic fatty liver disease (NAFLD)/Metabolic dysfunction-associated fatty liver disease (MAFLD)/Metabolic dysfunction-associated steatotic liver disease (MASLD)/Metabolic dysfunction-associated steatohepatitis (MASH), and gut-thyroid-microbiota interactions

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