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PMID: 19393193

Post-treatment assessments of cellular therapies also need to be enhanced

Research Square Preprint2024 Jan 24

Consistent with this, blocking either c-Maf or MafG induction during BDL protected against the fall in expression of GSH synthetic enzymes, GSH levels and BDL-induced liver injury [90]

Mechanisms of gastrointestinal microflora on drug metabolism in clinical practice

Confirming the significant role of Met35 of A1742 in these observations, when the Met35 residue was substituted by norleucine (as noted above, CH 2 in place of the S-atom of Met), no oxidative damage or neurotoxicity to hippocampal neurons was found (115)