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Nevertheless, even in the absence of glucose, albumin increased pathogenicity of 101, and an efg1 / cph1 deletion mutant (Supplementary Fig

Neurometabolite levels in the brains of patients with autism spectrum disorders: A meta-analysis of proton magnetic resonance spectroscopy studies (N = 1501)

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These should include: (1) Examination of these processes at different stages of the disease, (2) mechanistic studies to pinpoint signaling pathways involved in the regulation of the oxidative stress and DNA damage response markers and (3) studies addressing whether the deficits reported here can be reverted with antioxidant treatments