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In liver from SFG rats, the enhanced GSH level can also be explained by an increased amount of -GCS, a key enzyme in the biosynthesis of -glutamylcysteine and GSH
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Specifically, GPx1 deficiency augmented TNF suppression of the dual specific phosphatase DUSP4 to promote ERK1/2 activation, linking GPX1 and oxidant stress to MAPK regulation [6]
doi: 10.4103/2045-9912.279983
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