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Gomes FLT, Maranho RC, Tavares ER, et al

Its effects on tendon healing appear mediated through increased expression of growth hormone receptor and upregulation of tendon-specific markers

Philos Trans R Soc Lond B Biol Sci 2005, 360: 2355-2372

These effects are accompanied by reduced expression of proinflammatory cytokines (TNF- , IL-1, IL-6) and downregulation of RIP3, phosphorylated JNK, and c-Fos in the spinal cord, as well as an increased number of surviving neurons in the dorsal horn, indicating attenuation of CCI-induced neuronal death (121)