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METTL16 inhibits papillary thyroid cancer tumorigenicity through m6A/YTHDC2/SCD1-regulated lipid metabolism

It is caused by the accumulation of misfolded or unfolded proteins in the ER lumen, resulting in overexpression of ER stress markers such as growth arrest and DNA damage-inducible protein 153 (CCAAT/enhancer binding protein homologous protein), glucose-regulated protein 78 (GRP78), and C/-EBP homologous protein (CHOP)

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Some have robust clinical trial data