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Indirect antioxidant effects of NAC were demonstrated in a human ex vivo model of COPD exacerbation induced by lipopolysaccharide (LPS) [69, 70], and confirmed in patients with COPD who showed increased GSH levels in plasma and BAL after NAC treatment [71]

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To examine whether the administration of the Si-based agent alleviates the visceral pain and discomfort associated with UC, we investigated the neuronal activities in the nuclei of the dorsal medulla oblongata [the nuclei of the solitary tract (NST) and the dorsal vagal nuclei (DVN)], and the central amygdala (CeA) using immunostaining for c-Fos, a neuronal active marker

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