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Linear sequence motif analysis of the EGF-upregulated phosphorylation sites that were largely unaffected by the kinase inhibitor treatment revealed that both DDA and DIA could correctly identify the EGF-dependent but MEK-independent AKT kinase substrate motif RxRxx[s/t]

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This study shows, in a preclinical model, that MET inhibition can circumvent the recognized radioresistance of glioblastoma stem cells by enhancing the DDR

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