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10.1186/s13045-023-01424-6 J Hematol Oncol

Simmonds MC, Brown JV, Heirs MK, Higgins JP, Mannion RJ, Rodgers MA, et al

GBA gene mutations cause dysfunction of the autophagy-lysosome pathway and reduced glucocerebrosidase (GCase) activity [21], resulting in -syn aggregation and OS

Figure 3: Changes in the lameness period amongst the four experimental treatment groups

The body naturally synthesizes glutathione in nearly every cell, with particularly high concentrations in the liver

Within the spike, mutations tend to fall into four general classes, those that affect the receptor-binding domain (RBD), which are of importance because some may provide both immune escape or a fitness advantage, as well as facilitate reverse zoonotic events