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Data Availability No datasets were generated or analysed during the current study

doi: 10.1016/j.biomaterials.2011.07.071

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As a prime example, inflammation and oxidative stress, which normally emerge as a result of an abnormal pro-inflammatory response, or an exacerbated production of free radical species are increasingly recognized as the key abnormalities implicated in the development and acceleration of diabetes-linked complications (Randle et al., 1963) which stated that alteration in the uptake and metabolism of glucose and free fatty acids may be an instrumental process in the pathogenesis of insulin resistance, the major characteristic feature of T2D