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Pathologic findings reveal a correlation between the extent of nigrostriatal dopamine loss and the severity of certain PD motor features (e.g., bradykinesia)

There is also early work in a mouse muscle-injury model, where the inhibitor supported repair

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And if you had the MTHFR gene variant (which affects how your body processes B12 and folate, present in roughly 10-15% of the population), taking the wrong form of B12 may not address the underlying issue efficiently anyway