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The lack of a significant correlation between purine metabolites and 5-HIAA, suggests alterations in key 5-HT pathways that may both be modified by and contribute to oxidative stress via purine catabolism in FENNS
N-Methyl-D-aspartate receptor-mediated mitochondrial Ca2+ overload in acute excitotoxic motor neuron death: a mechanism distinct from chronic neurotoxicity after Ca2+ influx
Statements Author contributions XiL: Conceptualization, Data curation, Methodology, Validation, Visualization, Writing original draft, Writing review & editing
K.AringL.DasN
T., Munsterhjelm, E., Pyhi, R., Hynninen, M
