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Nuclear magnetic resonance (NMR) analysis suggested a weak interaction between -Syn and squalamine at the C -terminal, whereas the interaction between -Syn and the lipid vesicle took place at the N -terminal [293]

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It plays a central role in maintaining structural integrity across skin, vasculature, and connective tissue

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(2011) identified three mechanisms: BPC-157 increased fibroblast outgrowth from tendon explants in a dose-dependent manner, improved tendon cell survival under oxidative stress, and accelerated fibroblast migration through FAK-paxillin signaling