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p53 is known to have a dual role as a suppressor or promoter of ferroptosis (53,92)

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the release from the immune and epithelial cells of TNF-a is unregulated, and second, cells become progressively more sensitive to peroxidation damage as a result of continued TNF-a release, exacerbating local oxidative stress, which goes on to lessen available glutathione, often resulting in intensification of symptoms

However, these numbers do not capture the extent to which TBI affects those who do not seek immediate medical attention, those with masking comorbidities or poly-trauma and those with unclear mechanisms of injury [5]

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