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Though not yet proven, limited evidence suggests an increased cancer risk in those with higher-than-normal B12 levels

Abstract Background: The interplay between bacterial virulence factors and the host innate immune response in pneumococcal meningitis (PM) can result in uncontrolled neuroinflammation, which is known to induce apoptotic death of progenitor cells and post-mitotic neurons in the hippocampal dentate gyrus, resulting in cognitive impairment

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AOD-9604 completed Phase 2b human trials and failed its primary endpoint