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Kruse, T
Intestinal barrier dysfunction in inflammatory bowel disease: underpinning pathogenesis and therapeutics

In particular, the pathways involved in the metabolism of alanine, -alanine, aspartate, cysteine, histidine, glutamate, methionine, nitrogen, and nicotinate/nicotinamide, as well as the biosynthesis of acetyl-CoA are repressed

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found that 200 mg/kg of black ginseng extract inhibited -cell apoptosis and improved islet architecture, which led to the enhancement of -cell function and reduction of hyperglycemia in STZ-treated mice [30]
