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CD38 inhibition by apigenin ameliorates mitochondrial oxidative stress through restoration of the intracellular NAD+/NADH ratio and Sirt3 activity in renal tubular cells in diabetic rats

Key finding: NNMT positioned as a master metabolic regulator at the intersection of NAD+ salvage, one-carbon metabolism, and epigenetic control

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High expression of NNMT promotes proliferation and invasion in some tumors, while low expression of NNMT in some other tumors may be an adjustment measure for maintaining the specific tumor cell phenotype

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